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mig cxcl9 protein  (MedChemExpress)


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    MedChemExpress mig cxcl9 protein
    Mig Cxcl9 Protein, supplied by MedChemExpress, used in various techniques. Bioz Stars score: 94/100, based on 6 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/mig+cxcl9+protein/pmc12809752-82-0-6?v=MedChemExpress
    Average 94 stars, based on 6 article reviews
    mig cxcl9 protein - by Bioz Stars, 2026-07
    94/100 stars

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    <t>CXCL9</t> inhibits M2 macrophage-induced pro-tumor signaling in co-cultured A549 cells. (A) Representative Western blot bands showing the protein expression levels of VEGF-C, MMP2, MMP9, ERK, p-ERK, AKT, and p-AKT in A549 cells and M2 macrophages under different treatment conditions. β-actin was used as the loading control. (B-H) Quantitative analysis of relative protein expression levels normalized to β-actin. Data are presented as the mean ± standard deviation (n=3). (B) Relative expression of VEGF-C. (C) Relative expression of MMP2. (D) Relative expression of MMP9. (E) Relative expression of total ERK. (F) Relative expression of p-ERK. (G) Relative expression of total AKT. (H) Relative expression of p-AKT. *, indicates a significant difference (P<0.05).
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    <t>CXCL9</t> inhibits M2 macrophage-induced pro-tumor signaling in co-cultured A549 cells. (A) Representative Western blot bands showing the protein expression levels of VEGF-C, MMP2, MMP9, ERK, p-ERK, AKT, and p-AKT in A549 cells and M2 macrophages under different treatment conditions. β-actin was used as the loading control. (B-H) Quantitative analysis of relative protein expression levels normalized to β-actin. Data are presented as the mean ± standard deviation (n=3). (B) Relative expression of VEGF-C. (C) Relative expression of MMP2. (D) Relative expression of MMP9. (E) Relative expression of total ERK. (F) Relative expression of p-ERK. (G) Relative expression of total AKT. (H) Relative expression of p-AKT. *, indicates a significant difference (P<0.05).
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    CXCL9 inhibits M2 macrophage-induced pro-tumor signaling in co-cultured A549 cells. (A) Representative Western blot bands showing the protein expression levels of VEGF-C, MMP2, MMP9, ERK, p-ERK, AKT, and p-AKT in A549 cells and M2 macrophages under different treatment conditions. β-actin was used as the loading control. (B-H) Quantitative analysis of relative protein expression levels normalized to β-actin. Data are presented as the mean ± standard deviation (n=3). (B) Relative expression of VEGF-C. (C) Relative expression of MMP2. (D) Relative expression of MMP9. (E) Relative expression of total ERK. (F) Relative expression of p-ERK. (G) Relative expression of total AKT. (H) Relative expression of p-AKT. *, indicates a significant difference (P<0.05).

    Journal: Translational Cancer Research

    Article Title: The influence of CXCL9 on M2 macrophages in lung cancer development

    doi: 10.21037/tcr-2025-aw-2336

    Figure Lengend Snippet: CXCL9 inhibits M2 macrophage-induced pro-tumor signaling in co-cultured A549 cells. (A) Representative Western blot bands showing the protein expression levels of VEGF-C, MMP2, MMP9, ERK, p-ERK, AKT, and p-AKT in A549 cells and M2 macrophages under different treatment conditions. β-actin was used as the loading control. (B-H) Quantitative analysis of relative protein expression levels normalized to β-actin. Data are presented as the mean ± standard deviation (n=3). (B) Relative expression of VEGF-C. (C) Relative expression of MMP2. (D) Relative expression of MMP9. (E) Relative expression of total ERK. (F) Relative expression of p-ERK. (G) Relative expression of total AKT. (H) Relative expression of p-AKT. *, indicates a significant difference (P<0.05).

    Article Snippet: CXCL9 protein was acquired from MedChemExpress (HY-P7253).

    Techniques: Cell Culture, Western Blot, Expressing, Control, Standard Deviation